Halotestin 5 mg Fluoxymesterone 40 Tabs by Sterling Knight
Halotestin 5 mg Fluoxymesterone 40 Tabs by Sterling Knight
Halotestin is indicated for replacement therapy in conditions associated with deficiency or absence of endogenous testosterone such as primary hypogonadism, testicular failure, delayed puberty or metastatic mammary cancer in females.
40 tablets, each tablet contains 5mg Fluoxymesterone
hologram with serial NO and verification code
HALOTESTIN Tablets contain fluoxymesterone, an androgenic hormone.
Fluoxymesterone is a white or nearly white, odorless, crystalline powder, melting at or about 240° C, with some decomposition. It is practically insoluble in water, sparingly soluble in alcohol, and slightly soluble in chloroform.
The chemical name for fluoxymesterone is and roast-4-en-3-one, 9-fluoro-11,17- dihydroxy-17-methyl-, (11β,17β)-. The molecular formula is C20H29FO3 and the molecular weight 336.45.
The structural formula is represented below:
Halotestin® (fluoxymesterone) Structural Formula Illustration
Each HALOTESTIN (fluoxymesterone) tablet, for oral administration, contains 2 mg, 5 mg or 10 mg fluoxymesterone. Inactive ingredients: calcium stearate, corn starch, FD&C Yellow No. 5, lactose, sorbic acid, sucrose, tragacanth. In addition, the 2 mg tablet contains FD&C Yellow No. 6 and the 5 mg and 10 mg contain FD&C Blue No. 2.
Chemical structure: 9-alpha-fluoro-11-beta-hydroxy-17-alpha-methyl-4-androstene-3-one,17b-ol
The molecular weight of base: 336.4457
Effective dose: 20-30 mg / day orally
With the exception of perhaps Anadrol, Halotestin is the single most dangerous steroid to use. Its liver toxicity is unrivaled and you wouldn't be the first person to end up in the hospital with jaundice and dangerously elevated liver values after a hefty cycle of fluoxymesterone. My question has often been simply "Why?". Fluoxymesterone has a low anabolic capacity. The results in mass would be small to non-existent. Qualitatively similar gains as one would book with trenbolone, but tren would go for equal or less money, deliver three times the gains and wouldn't be half as risky to use. Therefor the sole marked use of fluoxymesterone that is actually warranted is that by power- and weightlifters seeking to boost strength while remaining in a set weight class.
In bodybuilding its used near the end of cutting cycles, since in people with an already low body-fat percentage it adds a distinct hardness and definition to the look, although, as stated, better and safer products will achieve similar effects. As with these alternatives, fluoxymesterone has absolutely zero estrogenic activity and will thus not add water or fat to the frame in any way.
While a definite increase in aggressiveness and a notable rise in erythropoiesis is noticed with the use of fluoxymesterone, it has been theorized that it actually has very moderate binding to the androgen receptor. Either that or it shows a higher affinity for other receptors. The enzyme aromatase comes to mind because of the effect it has, like a DHT compound would, on muscle hardness. The latter seems like a better explanation. On the one hand, there is nothing that would immediately indicate it acting on the androgen receptor, on the other, there is a very good likeness to other steroids that are mostly AR-mediated. It's my best guess that not all has been said about fluoxymesterone. It's not a very interesting or grateful object of study however due to the high risk and low yield of this particular steroid.
Athletes that may consider its use are endurance athletes that do not get drug tested (as it is quite easy to detect). The stimulating effect on erythropoiesis (red blood cell production) and cell respiration, such an athlete would find a good use for the increase in aerobic capacity noticed for this, without adding unnecessary bodyweight to the frame he has to carry. In this aspect, it may be good to note that a short cycle of Halotestin with a moderately long cycle of Equipoise may have some merit in this instance. Neither would increase water retention drastically, neither would give explosive gains. But both have positive effects on the VO2 max.
In any case, and whatever the reason of use, 4 weeks is the best duration of use, 6 weeks at the most. As stated before, many athletes, having used fluoxymesterone while not under supervision of a physician, have ended up in the hospital with life-threatening conditions.
Stacking and Use:
Halotestin is taken in mild doses (10-20 mg) every day for short periods of time, 4 weeks, 6 weeks at the very most due to its high level of toxicity. The use of anti-estrogens is not necessary since fluoxymesterone does not aromatize at all. As secondary drugs one may want to consider blood pressure medication such as catepressan to avoid hypertensive conditions. What you will definitely need is a check of liver values on a regular basis if you want to play it safe. I don't normally recommend the use of liver-protectors during a cycle as enhances liver function breaks down a greater amount of your steroid, but in this case you ought to make an exception. Milk thistle, dessicated liver, vitamin B6 and such both during and after a cycle are highly advised. There is no need for clomid of Nolvadex use after a cycle to bring back natural test.
Halotestin really only serves a purpose as a bodybuilding drug when the athlete is cutting. Probably in the late stages of a cutting cycle to promote muscle density and hardness, preserve muscle tissue and such. To that effect, it may be good to use some Halotestin (20-30 mg/day) the last 4 weeks of a boldenone or methenolone cycle for example, or at the end of a stack with trenbolone. It may make a good stacking partner for stanazolol (Winstrol/Stromba) as well since they serve the same purpose. But frankly in all cases opting for a higher dose of the other drug may be a better choice, both in terms of gains and safety. Boldenone (Equipoise) being the one possible exception. Due to its toxicity, Halotestin is not much sought out in stacks.
In the male—HALOTESTIN (fluoxymesterone) Tablets are indicated for:
Replacement therapy in conditions associated with symptoms of deficiency or absence of endogenous testosterone.
Primary hypogonadism (congenital or acquired)— testicular failure due to cryptorchidism, bilateral torsion, orchitis, vanishing testis syndrome; or orchidectomy.
Hypogonadotropic hypogonadism (congenital or acquired)—idiopathic gonadotropin or LHRH deficiency, or pituitary-hypothalamic injury from tumors, trauma, or radiation.
Delayed puberty, provided it has been definitely established as such, and is not just a familial trait.
In the female—HALOTESTIN (fluoxymesterone) Tablets are indicated for palliation of androgen-responsive recurrent mammary cancer in women who are more than one year but less than five years postmenopausal, or who have been proven to have a hormone-dependent tumor as shown by previous beneficial response to castration.
DOSAGE AND ADMINISTRATION
The dosage will vary depending upon the individual, the condition being treated, and its severity. The total daily oral dose may be administered singly or in divided (three or four) doses.
Male hypogonadism: For complete replacement in the hypogonadal male, a daily dose of 5 to 20 mg will suffice in the majority of patients. It is usually preferable to begin treatment with full therapeutic doses which are later adjusted to individual requirements. Priapism is indicative of excessive dosage and is an indication for the temporary withdrawal of the drug.
Delayed puberty: Dosage should be carefully titrated utilizing a low dose, appropriate skeletal monitoring, and by limiting the duration of therapy to four to six months.
Inoperable carcinoma of the breast in the female: The recommended total daily dose for palliative therapy in advanced inoperable carcinoma of the breast is 10 to 40 mg. Because of its short action, fluoxymesterone should be administered to patients in divided, rather than single, daily doses to ensure more stable blood levels. In general, it appears necessary to continue therapy for at least one month for a satisfactory subjective response, and for two to three months for an objective response.
Endocrine and urogenital
Female: the most common side effects of androgen therapy are amenorrhea and other menstrual irregularities; inhibition of gonadotropin secretion; and virilization, including deepening of the voice and clitoral enlargement. The latter usually is not reversible after androgens are discontinued. When administered to a pregnant woman, androgens can cause virilization of external genitalia of the female fetus.
Male: Gynecomastia, and excessive frequency and duration of penile erections. Oligospermia may occur at high dosage.
Skin and appendages
Hirsutism, male pattern of baldness, seborrhea, and acne.
Fluid and electrolyte disturbances
Retention of sodium, chloride, water, potassium, calcium, and inorganic phosphates.
Nausea, cholestatic jaundice, alterations in liver function tests, rarely hepatocellular neoplasms and peliosis hepatis (See WARNINGS).
Suppression of clotting factors II, V, VII, and X, bleeding in patients on concomitant anticoagulant therapy, and polycythemia.
Increased or decreased libido, headache, anxiety, depression, and generalized paresthesia.
Hypersensitivity, including skin manifestations and anaphylactoid reactions.
Drug Abuse And Dependence
Controlled Substance Class: Fluoxymesterone is a controlled substance under the Anabolic Steroids Control Act, and HALOTESTIN (fluoxymesterone) Tablets has been assigned to Schedule III.
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